Basic Medical Sciences for MRCP Part 1 by Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H

By Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H MPH

Written for applicants sitting their MRCP half 1 exam, this revision makes a speciality of the habitual subject matters which arise within the questions. The publication additionally features a bankruptcy on medical pharmacology (which by myself debts for as much as 30% of the questions), taking a look at facets of drug-induced disorder and drug interactions. eventually there's a bankruptcy on records and epidemiology which is never lined in different texts, yet is usually integrated within the exam.

  • Helps MRCP half 1 applicants organize for and go their exam.
  • Addresses an more and more vital subject within the exam.
  • Addresses a subject matter that's important to passing the examination, yet which such a lot applicants are poorly ready for.
  • Covers the entire proper uncomplicated technology topics plus contains medical pharmacology.
  • Is of use to applicants learning for different postgraduate tests corresponding to PLAB, USMLE and MRCPCH.
  • Is the 1st e-book of its variety within the club industry and is now considered as crucial for examination preparation.

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Removal of cells which have undergone DNA damage. )> z 0 ~ 0 r m c () r )> ::IJ ~ m 0 () z m Principal mediators p53 A tumour supressor gene which inhibits mitosis and drives apoptosis in cells in which DNA has been damaged. Mutation correlates with poor prognosis in tumours. bc/-2 A strong negative regulator of apoptosis. If it is overexpressed in tumours, cells have a prolonged survival. fas (CD95) A cellular receptor which, when activated, is directly coupled to the activation of intracellular proteases which lead to apoptosis.

Suis (iv) Br. 6 (Cont'd) - Genus and species Characteristics Diseases produced in humans Reservoirs of infection Pasteurella P multocida • Aerobic • Wound sepsis following a bite • Dogs and cats • Fastidious organism • Exotoxin • Demonstrated with difficulty in tissues, using a silver stain • Nosocomial pneumonia • Pontiac fever • Air-conditioning units • Pleomorphic • Capsulated • Tularaemia (ulceroglandular and oculoglandular form) (Fever, tonsillitis, headaches, hepatosplenomegaly, lvmphadenopathy) • Plague-like disease of rodents.

Abdominal wall defect. 3. g . previous affected chi ld. 4. Strong family history of chromosomal abnormality. 5. Carriers of X-linked recessive diseases who have decided to term inate male fetuses. 6. g. creati ne kinase in Duchenne muscu lar dystrophy, phenylalanine tolerance test in phenylketonuria, alphafetoprotein in neural tube defects. 7. g . a - and ~-thalassaemia, cystic fibrosis, fragile X syndrome, haemophilia A, Huntington 's disease, muscular dystrophy (Duchenne and Becker), myotonic dystrophy, spinal muscular atrophy.

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